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Clinical pharmacokinetics of carbamazepine

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It was previously reported that manipulations which prevent the hepatic recruitment of monocytes (i.e., Ccr2 knockout mice) inhibit the clearance of necrotic cell debris from the APAP-injured liver ( + monocytes in the livers of AALF mice coincided with a persistence of dead cell debris ( Figures 1 , 2 )

Sie sind im gesamten Krper vorhanden und spielen eine Rolle im Fettstoffwechsel sowie im Homocystein-Stoffwechsel